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In hematology, plasma cell dyscrasias (also termed plasma cell disorders and plasma cell proliferative diseases) are a spectrum of progressively more severe monoclonal gammopathies in which a clone or multiple clones of pre-malignant or malignant plasma cells (sometimes in association with lymphoplasmacytoid cells or B lymphocytes) over-produce and secrete into the blood stream a myeloma ...
Beginning in 1920, doctors at Johns Hopkins Hospital conducted research on sickle cell anemia, or sickle cell disease. Although their conclusions surrounding the disease are outdated, Doctors Taliaferro and Huck discovered a latent form of sickle cell anemia. Their study on sickle cell anemia was the first of many to occur at Hopkins.
Sickle cell anemia is a pleiotropic disease because the expression of a single mutated HBB gene produces numerous consequences throughout the body. The mutated hemoglobin forms polymers and clumps together causing the deoxygenated sickle red blood cells to assume the disfigured sickle shape. [32]
Peripheral blood smear in patient with thrombotic thrombocytopenic purpura. Typical schistocytes are annotated. A schistocyte or schizocyte (from Greek schistos for "divided" and kytos for "hollow" or "cell") is a fragmented part of a red blood cell.
This culminated in his most influential publication, "Immunologic Studies in Sickle Cell Anemia". [1] This work concluded that sickle cell anemia is most common among people of African heritage, that not all people with sickle cell anemia are anemic, that not all sickle cell cases are fatal, and that sickle cell anemia is a Mendelian trait. [6]
Ernest Edward Irons (February 17, 1877 – January 18, 1959) was an American physician who led several prominent medical organizations. Working as an intern under physician James B. Herrick, he identified abnormalities on a blood smear that resulted in the first published report of sickle cell disease.
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